A Diabetes Connection

Pertussis, Mumps, Rubella

The federal medical establishment – which would be the major source of funds for an investigation – is itself highly committed to the childhood vaccination program, which goes far to explain the absence of any official interest in this connection.

At least since the 1970s, PERTUSSIS vaccine has been known in animal experiments to stimulate over-production of insulin by the pancreas – followed by exhaustion and destruction of the islets cells with consequent under-production of insulin; in the first case the outcome is hypoglycemia, and in the latter it is diabetes. Physicians as early as 1949 called attention to low blood glucose in children who had severe reactions to the pertussis vaccine.

The MMR (measles, mumps, rubella) vaccine, especially its MUMPS and RUBELLA components, has been especially implicated in the causation of Type-I diabetes.

In the 1960’s and 1970’s, researchers came to realize that the effect of the rubella virus does not end at the moment of birth, but that it remains in the organism of the baby and continues to exert its influence for many years thereafter. Especially to be noted is the fact that up to 20 percent of these individuals later come down with Type-I diabetes. This may take from 5 to 20 years to develop, indicating that the rubella virus remains active in the organism for all that time.

“The mechanism of virus-induced diabetes is not known. Viruses associated with diabetes in animals may cause disease by (1) directly lysing [i.e., dissolving] the beta-cells; (2) triggering an autoimmune response; or (3) specifically impairing the secretory process of beta-cells through a persistent infection.” -E.J. Mayfield and colleagues.

He concluded that option (2) was the most probable one: the generation of an autoimmune state in which the body, as it were, becomes allergic to itself or to a part of itself.

Diabetes after a rubella vaccination probably represents a combined effect: the virus attacks the islet cells of the pancreas in an organism which has already been weakened by an autoimmune reaction to the same virus.